posters International Association for Breast Cancer Research 2014

Dysfunctional Aging Mammary Stem Cells (#108)

Lu-Zhe Sun 1 , Qiaoxiang Dong 1 2 , Hui Gao 1 2 , Danhan Wang 2 , Anqi Wu 2 , Abhik Bandyopadhyay 1 , Xiang Gu 1 , Yuanshuo Shi 2 , Changjiang Huang 2
  1. University of Texas Health Science Center at San Antonio, San Antonio, TEXAS, United States
  2. School of Environmental Sciences and Public Health, Wenzhou Medical University, Wenzhou, Zhejiang, China
Aging is the number one risk factor for breast cancer. Recent research implicated that mammary stem cells (MaSCs) and progenitors might initiate certain types of breast cancer.  In this study, MaSC-enriched basal cells were utilized for the evaluation of MaSC frequency and function during aging by in vitro mammosphere formation and 3D-ECM sphere differentiation assays and by in vivo cleared mammary fat pad transplantation (IVT) as we reported recently (Dong et al., Stem Cell Res. 10:396-404, 2013).  We found that the basal-to-luminal cell ratios and the frequency of MaSCs analyzed with the in vitro assays increased steadily with increasing age in various strains of mice. The old (25-32 months) mammary gland had significantly higher frequency of atypical ductal hyperplasia (ADH) and ductal carcinoma in situ (DCIS) lesions in comparison with the young (2-4 months) mammary gland. Subsequent IVT using mammospheres or 3D-ECM structures formed by young or old  MaSCs showed that the regenerated glands from old MaSCs had significantly higher number of spontaneous ADH/DCIS lesions than those from young MaSCs. These findings indicate that aged MaSCs can serve as the cell of origin for early neoplastic transformation in breast tissue. Subsequent whole genome RNA-seq revealed age-associated differential expression of genes involved in immune, inflammatory, and wounding responses in both mammosphere-forming cells and stromal cells suggesting that these may be the main cellular processes contributing to the dysfunctional MaSC phenotypes. Consistently, treatment of old mice with rapamycin, an anti-inflammation drug, showed that both 5-10 days or 2 year treatment reversed phenotypic changes associated with aged mammary gland. Histological analysis of regenerated glands by aged MaSCs from control and rapamycin-treated mice showed significant decreases of early neoplastic lesions in rapamycin-treated group.  In conclusion, our findings suggest that aging causes MaSC to form early neoplastic lesions, which can be inhibited by rapamycin treatment.